ENTDr. Trisha SrivastavaObstructive Sleep Apnoea

Consultant, ENT, Artemis Hospitals, Gurugram, India

Part 2 of 8 in The Science Behind Snoring

Three Patients, One Symptom: The Clinical Phenotypes of Snoring

May 17, 2026

Dr. Srivastava opens with three real patient profiles to show how one symptom can mask entirely different mechanisms. A 42-year-old man with a sedentary lifestyle and a BMI of 34 sleeps 7 to 8 hours but wakes unrefreshed, with brain fog and daytime irritability; examination shows obesity, a thick neck, and a bulky tongue and palate. A 28-year-old athletic man with normal BMI has chronic nasal allergies that block his nose whenever he lies down, forcing mouth breathing and snoring. A 35-year-old woman with normal BMI and no nasal complaints instead has a sensitive throat that flares with weather change or cold and sour foods, waking sore but improving through the day.

Her core teaching point: not every snoring presentation should be branded obstructive sleep apnoea. Any blockage from the nasal vestibule to the level of the vocal cord, such as a deviated septum, turbinate hypertrophy, adenoids, tonsillar hypertrophy, or a tongue that falls back during sleep, can produce snoring or fragmented sleep, and the cause has to be identified at the correct anatomical level before treatment is planned.

This guide is based on a live Jivo Masterclass — Dr. Trisha Srivastava taught doctors across Africa on May 17, 2026.

FROM THE LIVE Q&A

DR

Dr. Isaya, Tanzania

Can you elaborate more on circadian rhythm and hormones in relation to sleep?

TS

Dr. Trisha Srivastava

Stage 3 of non-REM sleep is when growth hormones are released, responsible for repairing the day's muscle wear and tear and, in children, for height and weight gain. If a patient isn't reaching stage 3 and stays mostly in light sleep, that reparative action doesn't happen and they feel tired the next day — in children, the consequence can be a genuine failure to grow as expected despite eating well.

See all 9 questions from this masterclass →

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Frequently Asked Questions

Can you elaborate more on the stages of sleep?

Every individual goes through 4 to 5 sleep cycles a night, each around 90 to 110 minutes, split into non-REM and REM sleep. Non-REM has three stages: N1 is the lightest — waking from it causes no brain fog; N2 and N3 together make up around 75% of total sleep, and it's in N3 that physical recovery, growth hormone release and cardiovascular recovery happen. REM follows, the dreaming stage, where memories are consolidated into long-term storage.

How do patients find their way to your clinic for this problem?

It's a blend — mostly patients I've already treated referring others after a good outcome, some general awareness through social media, cross-referrals from other departments within Artemis, and GP referrals from outside the hospital, which make up roughly 20 to 30% of this OPD. GPs do a great job fixing the simple presentations themselves; it's when the problem isn't resolving that they refer on.

Can taking wine at night help reduce snoring?

I haven't read that in any textbook or literature. Chamomile tea is said to calm the nerves and may help, and melatonin helps at night, but wine and alcohol generally disrupt sleep rather than improve it — alcohol causes dryness, and people often don't eat adequately alongside it, leading to a calorie deficit. If anything, it disrupts sleep rather than making it better.

Can you compare CPAP and BiPAP?

A PAP machine is essentially an airway splint that sends a puff of air to open the blockage at the retropalatal or base-of-tongue level. CPAP delivers one continuous fixed pressure through both inspiration and expiration. BiPAP sets different pressures for each phase — for example 8 cm H2O on inspiration but only 4 cm H2O on expiration — which many patients who can't tolerate CPAP's constant pressure find more comfortable to breathe against.

I've observed athletes complaining of lack of sleep after vigorous exercise. What is the relationship between vigorous exercise and poor sleep?

It could be related to the significant muscle wear and tear from extensive exercise, with the recovery process possibly interfering with sleep — but I have to be honest that I'm not certain about this specifically in athletes, and it would need further looking into.

Can two patients with identical snoring symptoms have completely different causes?

Yes. A 42-year-old man with obesity and a thick neck, a 28-year-old athletic man with nasal allergies, and a 35-year-old woman with a sensitive throat all present with snoring and poor sleep, yet each has a distinct underlying mechanism.

Does a normal BMI rule out snoring caused by an obstruction?

No. Both the athletic 28-year-old man and the 35-year-old woman in these profiles have a normal BMI, but his snoring traces to nasal allergies and turbinate hypertrophy while hers traces to throat sensitivity and mild oropharyngeal crowding.

Should every snoring presentation be labelled obstructive sleep apnoea?

No. Any obstruction from the nasal vestibule to the level of the vocal cord, such as a deviated septum, turbinate hypertrophy, adenoids, tonsillar hypertrophy, or a tongue that falls back during sleep, can cause snoring or fragmented sleep. The cause has to be identified at the correct anatomical level before treatment is planned.

What examination findings distinguish an obesity driven snoring case from an allergy driven one?

The obesity driven case shows a thick neck with a bulky tongue and palate. The allergy driven case shows a deviated septum with turbinate hypertrophy and a nose that blocks specifically when lying down.

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